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Transformed DLBCL routinely include CDKN2A deletions and MYC translocations or amplifications in addition to the genomic alterations presently current in the first CLL, but deficiency the typical mutations observed in Principal DLBCL indicating they may perhaps correspond to a special biological group.80 Richter transformation also occurs in clients addressed with BTK inhibitors. These tumors do not typically get BTK or PLCG2 mutations but, if these have been current in the initial CLL, subclones could emerge with extra independent mutations.89,ninety